metabolic resistance
Metribuzin resistance via enhanced metabolism in a multiple herbicide resistant Lolium rigidum population
Most Australian crop farm populations of the grass weed Lolium rigidum are multiple herbicide-resistant. Most resistant populations exhibit target site mutations (e.g. ACCase, ALS), as well as metabolic resistance due to cytochrome P450, catalysed by enhanced rates of herbicide metabolism.
Widespread occurrence of both metabolic and target-site herbicide resistance mechanisms in Lolium rigidum populations
Lolium rigidum populations in Australia and globally have demonstrated rapid and widespread evolution of resistance to acetyl coenzyme A carboxylase (ACCase)-inhibiting and acetolactate synthase (ALS)-inhibiting herbicides. Thirty-three resistant L. rigidum populations, randomly collected from crop fields in a most recent resistance survey, were analysed for non-target-site diclofop metabolism and all known target-site ACCase gene resistance-endowing mutations.